Where is striated skeletal muscle found




















Heart failure. N Engl J Med. Genetics of congenital heart disease: the glass half empty. Circulation research. Hanson J, Huxley HE. Structural basis of the cross-striations in muscle. Huxley HE. Electron microscope studies of the organisation of the filaments in striated muscle. Biochim Biophys Acta. Luther PK. The vertebrate muscle Z-disc: sarcomere anchor for structure and signalling. J Muscle Res Cell Motil. The sarcomeric Z-disc and Z-discopathies.

J Biomed Biotechnol. Al-Qusairi L, Laporte J. T-tubule biogenesis and triad formation in skeletal muscle and implication in human diseases. Fill M, Copello JA. Ryanodine receptor calcium release channels. Schiaffino S, Margreth A. Coordinated development of the sarcoplasmic reticulum and T system during postnatal differentiation of rat skeletal muscle. J Cell Biol.

Discrimination between fast- and slow-twitch fibres of guinea pig skeletal muscle using the relative surface density of junctional transverse tubule membrane. Sawada K, Kawamura K. Architecture of myocardial cells in human cardiac ventricles with concentric and eccentric hypertrophy as demonstrated by quantitative scanning electron microscopy. Heart and vessels. Cardiomyocyte proliferation contributes to heart growth in young humans.

Cardiomyocyte DNA synthesis and binucleation during murine development. The American journal of physiology. Venable JH. Constant cell populations in normal, testosterone-deprived and testosterone-stimulated levator ani muscles.

Am J Anat. Enesco M, Puddy D. Determination of cell types and numbers during cardiac development in the neonatal and adult rat and mouse. American journal of physiology Heart and circulatory physiology. Pressure overload induces early morphological changes in the heart. The American journal of pathology.

Revisiting Cardiac Cellular Composition. Coupling of cardiac electrical activity over extended distances by fibroblasts of cardiac origin. Electrotonic loading of anisotropic cardiac monolayers by unexcitable cells depends on connexin type and expression level. American journal of physiology Cell physiology. Rohr S. Arrhythmogenic implications of fibroblast-myocyte interactions.

Circulation Arrhythmia and electrophysiology. Ongstad E, Kohl P. Fibroblast-myocyte coupling in the heart: Potential relevance for therapeutic interventions. J Mol Cell Cardiol. Dual effect of oxygen on magnitude and uniformity of coronary intercapillary distance. Korthuis RJ. Skeletal Muscle Circulation. Andersen P, Kroese AJ. Capillary supply in soleus and gastrocnemius muscles of man.

Pflugers Arch. Skeletal muscle capillary geometry: adaptation to chronic hypoxia. Respir Physiol. Analysis of capillary geometry in rat subepicardium and subendocardium. Am J Physiol. Layland J, Kentish JC. Excitability and inhibitability of motoneurons of different sizes. J Neurophysiol. Huxley AF. Muscle structure and theories of contraction. Prog Biophys Biophys Chem. The cellular basis of the length-tension relation in cardiac muscle. Passive tension in cardiac muscle: contribution of collagen, titin, microtubules, and intermediate filaments.

Biophysical journal. Mauro A. Satellite cell of skeletal muscle fibers. J Biophys Biochem Cytol. An absolute requirement for Pax7-positive satellite cells in acute injury-induced skeletal muscle regeneration. Embryonic founders of adult muscle stem cells are primed by the determination gene Mrf4. Dev Biol. Myf5 expression during fetal myogenesis defines the developmental progenitors of adult satellite cells.

Asymmetric self-renewal and commitment of satellite stem cells in muscle. Cossu G, Tajbakhsh S. Oriented cell divisions and muscle satellite cell heterogeneity. Myf5-positive satellite cells contribute to Pax7-dependent long-term maintenance of adult muscle stem cells.

Cell Stem Cell. Cellular and molecular regulation of muscle regeneration. Satellite cells and the muscle stem cell niche. Origin of cardiomyocytes in the adult heart. Circ Res. Inflammatory monocytes recruited after skeletal muscle injury switch into antiinflammatory macrophages to support myogenesis.

J Exp Med. Madaro L, Bouche M. From innate to adaptive immune response in muscular dystrophies and skeletal muscle regeneration: the role of lymphocytes. Biomed Res Int. Immune modulation of stem cells and regeneration. The FEBS journal. Nat Cell Biol. Building and re-building the heart by cardiomyocyte proliferation. Mending broken hearts: cardiac development as a basis for adult heart regeneration and repair. Nat Rev Mol Cell Biol. Macrophages are required for neonatal heart regeneration. The Journal of clinical investigation.

Sutton MG, Sharpe N. Left ventricular remodeling after myocardial infarction: pathophysiology and therapy. Salamander limb regeneration involves the activation of a multipotent skeletal muscle satellite cell population. Tales of regeneration in zebrafish. Dev Dyn.

Regeneration of skeletal muscle. Cell Tissue Res. The healthcare costs of sarcopenia in the United States. J Am Geriatr Soc. Sarcopenia: prevalence, mechanisms, and functional consequences. Interdiscip Top Gerontol. Cellular and molecular mechanisms underlying age-related skeletal muscle wasting and weakness.

In vivo physiological cross-sectional area and specific force are reduced in the gastrocnemius of elderly men. J Appl Physiol ; 99 — Longitudinal muscle strength changes in older adults: influence of muscle mass, physical activity, and health. Aging of human muscle: structure, function and adaptability. Scand J Med Sci Sports. Rejuvenation of the muscle stem cell population restores strength to injured aged muscles.

Nat Med. CDC National Health Report: leading causes of morbidity and mortality and associated behavioral risk and protective factors--United States, — Morbidity and mortality weekly report Surveillance summaries. Diagnosis and management of diastolic dysfunction and heart failure. American family physician.

Racial differences in infant mortality attributable to birth defects in the United States, — Birth defects research Part A, Clinical and molecular teratology. Hoffman JI, Kaplan S. The incidence of congenital heart disease. Journal of the American College of Cardiology. Stem cell therapy and tissue engineering for correction of congenital heart disease. Frontiers in Cell and Developmental Biology. Mercuri E, Muntoni F.

Muscular dystrophy: new challenges and review of the current clinical trials. Curr Opin Pediatr. The dystrophin glycoprotein complex: signaling strength and integrity for the sarcolemma. Muscle fat-fraction and mapping in Duchenne muscular dystrophy: evaluation of disease distribution and correlation with clinical assessments.

Preliminary experience. Skeletal Radiol. Absence of extraocular muscle pathology in Duchenne's muscular dystrophy: role for calcium homeostasis in extraocular muscle sparing. Mechanisms of muscle weakness in muscular dystrophy.

The Journal of general physiology. T2 mapping in Duchenne muscular dystrophy: distribution of disease activity and correlation with clinical assessments. Muscle histology vs MRI in Duchenne muscular dystrophy. Muscle diseases: the muscular dystrophies. Annu Rev Pathol. Myocardial involvement is very frequent among patients affected with subclinical Becker's muscular dystrophy.

Cardiac involvement in patients with muscular dystrophies: magnetic resonance imaging phenotype and genotypic considerations. Circulation Cardiovascular imaging. Myoblast transfer therapy: is there any light at the end of the tunnel? Direct isolation of satellite cells for skeletal muscle regeneration. Primary rat muscle progenitor cells have decreased proliferation and myotube formation during passages. Cell Prolif. Dynamics of myoblast transplantation reveal a discrete minority of precursors with stem cell-like properties as the myogenic source.

Slow-dividing satellite cells retain long-term self-renewal ability in adult muscle. J Cell Sci. Myotubes differentiate optimally on substrates with tissue-like stiffness: pathological implications for soft or stiff microenvironments.

Substrate elasticity regulates skeletal muscle stem cell self-renewal in culture. Mol Ther. Derivation of engraftable skeletal myoblasts from human embryonic stem cells. Stem Cells. Stem cell reviews. Myogenic differentiation of muscular dystrophy-specific induced pluripotent stem cells for use in drug discovery.

Stem cells translational medicine. Epigenetic reprogramming of human embryonic stem cells into skeletal muscle cells and generation of contractile myospheres. Cell Rep. Myoblasts derived from normal hESCs and dystrophic hiPSCs efficiently fuse with existing muscle fibers following transplantation.

Derivation and expansion of PAX7-positive muscle progenitors from human and mouse embryonic stem cells. Stem Cell Reports. A zebrafish embryo culture system defines factors that promote vertebrate myogenesis across species.

Differentiation of pluripotent stem cells to muscle fiber to model Duchenne muscular dystrophy. Nat Biotechnol. Pw1, a novel zinc finger gene implicated in the myogenic and neuronal lineages.

Identification and characterization of a non-satellite cell muscle resident progenitor during postnatal development. Cell Death Dis. Mesenchymal progenitors distinct from satellite cells contribute to ectopic fat cell formation in skeletal muscle.

Fibroadipogenic progenitors mediate the ability of HDAC inhibitors to promote regeneration in dystrophic muscles of young, but not old Mdx mice. The extraocular muscle stem cell niche is resistant to ageing and disease. Front Aging Neurosci. Isolation and characterization of mesoangioblasts from mouse, dog, and human tissues. Curr Protoc Stem Cell Biol. The meso-angioblast: a multipotent, self-renewing cell that originates from the dorsal aorta and differentiates into most mesodermal tissues.

Skeletal myogenic progenitors originating from embryonic dorsal aorta coexpress endothelial and myogenic markers and contribute to postnatal muscle growth and regeneration. Skeletal muscle pericyte subtypes differ in their differentiation potential.

Stem Cell Res. Pericytes of human skeletal muscle are myogenic precursors distinct from satellite cells. A perivascular origin for mesenchymal stem cells in multiple human organs. Tedesco FS, Cossu G. Stem cell therapies for muscle disorders. Curr Opin Neurol. Transplantation of genetically corrected human iPSC-derived progenitors in mice with limb-girdle muscular dystrophy. Sci Transl Med. Mesoangioblast stem cells ameliorate muscle function in dystrophic dogs.

Cell therapy of alpha-sarcoglycan null dystrophic mice through intra-arterial delivery of mesoangioblasts. Mesodermal iPSC-derived progenitor cells functionally regenerate cardiac and skeletal muscle.

Nat Commun. Cardiac tissue engineering and regeneration using cell-based therapy. Stem cells and cloning : advances and applications. Stem cells international. Myoblast transplantation for heart failure. Autologous skeletal myoblast transplantation for the treatment of postinfarction myocardial injury: phase I clinical study with 12 months of follow-up. Am Heart J. Skeletal myoblast transplantation in ischemic heart failure: long-term follow-up of the first phase I cohort of patients. Final results of a phase IIa, randomised, open-label trial to evaluate the percutaneous intramyocardial transplantation of autologous skeletal myoblasts in congestive heart failure patients: the SEISMIC trial.

Reinecke H, Murry CE. Transmural replacement of myocardium after skeletal myoblast grafting into the heart. Too much of a good thing? Cardiovasc Pathol. Skeletal myoblasts preserve remote matrix architecture and global function when implanted early or late after coronary ligation into infarcted or remote myocardium.

Donor cell-type specific paracrine effects of cell transplantation for post-infarction heart failure. Dimmeler S, Zeiher AM. Cell therapy of acute myocardial infarction: open questions. Minimal criteria for defining multipotent mesenchymal stromal cells. The International Society for Cellular Therapy position statement. Concise review: Adipose-derived stem cells as a novel tool for future regenerative medicine. Adult bone marrow cell therapy improves survival and induces long-term improvement in cardiac parameters: a systematic review and meta-analysis.

The effect of bone marrow mononuclear stem cell therapy on left ventricular function and myocardial perfusion. Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology. Intracoronary stem cell infusion after acute myocardial infarction: a meta-analysis and update on clinical trials. Under the light microscope, muscle cells appear striated with many nuclei squeezed along the membranes. The striation is due to the regular alternation of the contractile proteins actin and myosin, along with the structural proteins that couple the contractile proteins to connective tissues.

The cells are multinucleated as a result of the fusion of the many myoblasts that fuse to form each long muscle fiber. Cardiac muscle forms the contractile walls of the heart. The cells of cardiac muscle, known as cardiomyocytes, also appear striated under the microscope. Unlike skeletal muscle fibers, cardiomyocytes are single cells with a single centrally located nucleus. A principal characteristic of cardiomyocytes is that they contract on their own intrinsic rhythm without external stimulation.

Cardiomyocytes attach to one another with specialized cell junctions called intercalated discs. Intercalated discs have both anchoring junctions and gap junctions. Attached cells form long, branching cardiac muscle fibers that act as a syncytium, allowing the cells to synchronize their actions.

The cardiac muscle pumps blood through the body and is under involuntary control. Smooth muscle tissue contraction is responsible for involuntary movements in the internal organs. It forms the contractile component of the digestive, urinary, and reproductive systems as well as the airways and blood vessels. Each cell is spindle shaped with a single nucleus and no visible striations Figure 4. Watch this video to learn more about muscle tissue.

In looking through a microscope how could you distinguish skeletal muscle tissue from smooth muscle? Intercalated discs have both anchoring junctions and gap junctions.

Attached cells form long, branching cardiac muscle fibers that are, essentially, a mechanical and electrochemical syncytium allowing the cells to synchronize their actions. The cardiac muscle pumps blood through the body and is under involuntary control. The attachment junctions hold adjacent cells together across the dynamic pressures changes of the cardiac cycle.

Smooth muscle tissue contraction is responsible for involuntary movements in the internal organs. It forms the contractile component of the digestive, urinary, and reproductive systems as well as the airways and arteries. Each cell is spindle shaped with a single nucleus and no visible striations.

It is responsible for voluntary movement, produces heat, and protects organs. It is attached to bones and around entrance points to body. It contracts to pump blood and is found only in the heart. It is responsible for involuntary movement e. It is found on the walls of major organs and passageways. Striated muscle : Muscle tissue in which the contractile fibrils in the cells are aligned in parallel bundles, so that their different regions form stripes visible in a microscope.

Myocyte : A muscle cell. Striation : Muscle tissue in which the contractile fibrils in the cells are aligned in parallel bundles, so that their different regions form stripes visible in a microscope. Cardiac muscle myocardium : Involuntary, striated muscle that constitutes the main tissue of the walls of the heart.

Smooth muscle : Muscle tissue in which the contractile fibrils are not highly ordered, occurring in the gut and other internal organs and not under voluntary control. Next Trial Session:. Recorded Trial Session. This is a recorded trial for students who missed the last live session. Waiting List Details:. Due to high demand and limited spots there is a waiting list. What is the difference between skeletal muscle and muscle mass? Muscle mass indicates the weight of muscle in your body.

Muscle mass is composed of 3 types of muscles : skeletal , smooth, and cardiac muscle. Skeletal muscle is also called striated muscle and is under voluntary control. Cardiac muscle is a mix between skeletal and smooth muscle : it is an involuntary striated muscle. How does a skeletal muscle work?

Together, the skeletal muscles work with your bones to give your body power and strength. In most cases, a skeletal muscle is attached to one end of a bone. It stretches all the way across a joint the place where two bones meet and then attaches again to another bone. Can you build skeletal muscle? This is because of the three major muscle types — cardiac, smooth, and skeletal — skeletal muscle mass is the only type of muscle that you can actively grow and develop through proper exercise and nutrition.

What is another name for smooth muscles? Smooth muscle , also called involuntary muscle , muscle that shows no cross stripes under microscopic magnification. It consists of narrow spindle-shaped cells with a single, centrally located nucleus. Smooth muscle tissue, unlike striated muscle , contracts slowly and automatically. What do skeletal muscles look like? Skeletal muscle looks striped or "striated" — the fibres contain alternating light and dark bands striations like horizontal stripes on a rugby shirt.



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